Food Related Brain Activation in patients with Psychological Disorders
Mojtaba Barzegar1*, Qurat Ul Ain Muhammad2* and Beyzanur Ceran3
11Associate Medical Physicist, Radiation Oncology department, National Center for
Cancer Care and Research (NCCCR), Hamad Medical Corporation, Doha, Qatar
2Bachelor of Medicine, Bachelor of Surgery (MBBS), Rawalpindi Medical University, Rawalpindi, Pakistan
3Medical Student, Ankara Yildirim Beyazit University, Ankara, Turkey
*Corresponding Author: Mojtaba Barzegar, Associate Medical Physicist,
Radiation Oncology department, National Center for Cancer Care and Research (NCCCR), Hamad Medical Corporation, Doha, Qatar.
Received:
March 16, 2023; Published: April 01, 2023
Abstract
Food-related brain activation is a term used for the alterations in brain activity that occurs in response to food cues, such as the sight or smell of food. Whenever a person sees, smells, or tastes food, a complex network of neural circuits is stimulated; this releases neurotransmitters such as dopamine and opioids [1]. These neural circuits are interconnections between several areas of the brain that are involved in processing food cues such as the prefrontal cortex, insula, amygdala, hippocampus, and striatum [2].
References
- Stice Eric., et al. “Relation of reward from food intake and anticipated food intake to obesity: a functional magnetic resonance imaging study”. Journal of Abnormal Psychology4 (2008): 924.
- Buckner SL., et al. “Lower extremity strength, systemic inflammation and all-Cause mortality: application to the “fat but fit” paradigm using cross-sectional and longitudinal designs”. Physiology and Behavior 149 (2015): 199-202.
- Lassale C., et al. “Healthy dietary indices and risk of depressive outcomes: a systematic review and meta-analysis of observational studies”. Molecular Psychiatry 24 (2019): 965-986.
- Salari-Moghaddam A., et al. “Glycemic index, glycemic load, and depression: a systematic review and meta-analysis”. European Journal of Clinical Nutrition 73 (2019): 356-365.
- Noble EE., et al. “Gut to brain dysbiosis: mechanisms linking western diet consumption, the microbiome, and cognitive impairment”. Frontiers in Behavioral Neuroscience 11 (2017): 9.
- Yuan N., et al. “Inflammation-related biomarkers in major psychiatric disorders: a cross-disorder assessment of reproducibility and specificity in 43 meta-analyses”. Translational Psychiatry 9 (2019): 233.
- Firth J., et al. “Diet as a hot topic in psychiatry: a population-scale study of nutritional intake and inflammatory potential in severe mental illness”. World Psychiatry 17 (2018): 365-367.
- Miller EK., et al. “What we can do and what we cannot do with fMRI”. Nature Neuroscience7197 (2008): 869-878.
- Babakhani B., et al. “A Preliminary Study of the Efficacy of Transcranial Direct Current Stimulation in Trigeminal Neuralgia”. Frontiers in Human Neuroscience16 (2022): 848347.
- Volkow Nora D., et al. “Overlapping neuronal circuits in addiction and obesity: evidence of systems pathology”. Philosophical Transactions of the Royal Society B: Biological Sciences1507 (2008): 3191-3200.
- Oberndorfer Tyson A., et al. “Altered insula response to sweet taste processing after recovery from anorexia and bulimia nervosa”. American Journal of Psychiatry10 (2013): 1143-1151.
- Stice E., et al. “Relative ability of fat and sugar tastes to activate reward, gustatory, and somatosensory regions”. American Journal of Clinical Nutrition4 (2013): 995-1005.
- Momenian Mohammad., et al. “Compound words are decomposed regardless of semantic transparency and grammatical class: An fMRI study in Persian”. Lingua 259 (2021): 103120.
- Young Kymberly D., et al. “Real-time FMRI neurofeedback training of amygdala activity in patients with major depressive disorder”. PloS One2 (2014): e88785.
- Bhavsar Y., et al. “Artificial intelligence and stochastic process-based analysis of human psychiatric disorders”. JAMSAT 1 (2021): 33-53.
Citation
Copyright