Acta Scientific Ophthalmology (ISSN: 2582-3191)

Editorial Volume 5 Issue 2

Bisphosphonates and Ocular Inflammation

Ali Nouraeinejad1,2*

1Ph.D. in Clinical Ophthalmology, Tehran University of Medical Sciences, Tehran, Iran
2University College London (UCL), London, United Kingdom

*Corresponding Author: Ali Nouraeinejad, Ph.D. in Clinical Ophthalmology, Tehran University of Medical Sciences, Tehran, Iran.

Received: November 24, 2021; Published: January 01, 2022

Abstract

Osteoporosis, as a musculoskeletal disease, is assigned by low bone mass and degradation of bone microarchitecture, culminating in an escalation in bone weakness and propensity to fracture [1,2]. When the cumulative effect of bone resorption is in excess of bone formation, osteoporosis is induced [1]. Osteoporotic fractures are linked to substantial morbidity, mortality, and high healthcare expenses [1].

References

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  19. Peterson JD and Bedrossian EH. “Bisphosphonate-associated orbital inflammation—a case report and review”. Orbit 31 (2012): 119-123.
  20. Hewitt RE., et al. “The bisphosphonate acute phase response: rapid and copious production of proinflammatory cytokines by peripheral blood gd T cells in response to aminobisphosphonates is inhibited by statins”. Clinical and Experimental Immunology 1 (2005): 101-111.
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Citation

Citation: Ali Nouraeinejad. “Bisphosphonates and Ocular Inflammation". Acta Scientific Ophthalmology 5.2 (2022): 01-03.

Copyright

Copyright: © 2022 Ali Nouraeinejad. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.




Metrics

Acceptance rate35%
Acceptance to publication20-30 days
ISI- IF1.042
JCR- IF0.24

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