Kunal Joon*

University of NIIMS, Uttar Pradesh, India

*Corresponding Author: Kunal Joon, University of NIIMS, Uttar Pradesh, India.

Received: May 24, 2024; Published: June 01, 2024

Abstract

Alzheimer’s disease (AD), the leading cause of dementia worldwide, is characterized by the accumulation of the β-amyloid peptide (Aβ) within the brain along with hyperphosphorylated and cleaved forms. Accumulation of cerebral amyloid-beta peptide (Abeta) is essential for developing synaptic and cognitive deficits in Alzheimer’s disease.

 Keywords: Neurodegeneration; Dementia; BACE1; α-Secretase; γ-Secretase; Aging

References

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Citation

Citation: Kunal Joon. “Amyloid Precursor Protein Processing and Alzheimer’s Disease”. Acta Scientific Neurology 7.7 (2024): 01-02.

Copyright

Copyright: © 2024 Kunal Joon. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.