Redha Saleh El-Ramli*

Consultant Gastroenterologist, Al-Safwa General Hospital, Benghazi and Senior Lecturer at Tobruk University, Faculty of Medicine

*Corresponding Author: Redha Saleh El-Ramli, Consultant Gastroenterologist, Al-Safwa General Hospital, Benghazi and Senior Lecturer at Tobruk University, Faculty of Medicine.

Received: February 25, 2025; Published: March 08, 2025

Abstract

Polycystic ovary syndrome remains the commonest endocrine dysfunction amongst women of childbearing age, however it is diagnosis and treatment is one of the predicament issues to Gynaecologist and endocrinologist [1]. It is characterised by cystic tissue, oligo/amenorrhoea, hyperandrogenism, anovulatory infertility and metabolic syndrome [1,2]. It has prevalence between 5 to 21% based on ethnic groups, study population’s characteristics and varies approach to identify hallmarks of PCOS.

References

1. Shouzhen Chen., et al. “Calprotectin is a potential prognostic marker for polycystic ovary syndrome”. Annals of Clinical Biochemistry: International Journal of Laboratory Medicine2 (2016).
2. Bihter Senem Feyzioglu and Zerrin Avul. “Evaluation of the relationship between polycystic ovary syndrome and intestinal inflammation as measured by fecal calprotectin levels”. Zeynep Kamil Medical Journal 4 (2024): 207-212.
3. Necati Hancerliogullari., et al. “Serum calprotectin levels as markers of inflammation, insulin resistance and hyperandrogenism in women with polycystic ovary syndrome”. Medicine (Baltimore) 51 (2022): e32326.
4. Marzieh Saei Ghare Naz., et al. “Polycystic ovary syndrome and irritable bowel syndrome: Is there a common pathway? Endocrinology”. Diabetes Metabolism2 (2024): e00477.

Citation

Citation: Redha Saleh El-Ramli. “Raised Faecal Calprotectin in Polycystic Ovary Syndrome: is it a Marker of Inflammatory Bowel Disease?".Acta Scientific Gastrointestinal Disorders 8.4 (2025): 09-10.

Copyright

Copyright: © 2025 Redha Saleh El-Ramli. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.