Sheikh Uzma Farooq1, Aditya Sharma2*, Sumaiya Bashir Khanday3, Parth Sharma4 and Janvi4
1Department of Veterinary Pharmacology and Toxicology, Khalsa College of Veterinary and Animal Sciences, Amritsar, India
2Department of Veterinary Pathology, Khalsa College of Veterinary and Animal Sciences, Amritsar, India
3Department of Veterinary Physiology and Biochemistry, Khalsa College of Veterinary and Animals Sciences, Amritsar, India
43rd Professional Year Student, KCVAS, Amritsar, India
*Corresponding Author: Aditya Sharma, Department of Veterinary Pathology, Khalsa College of Veterinary and Animal Sciences, Amritsar, India.
Received: November 07, 2022; Published: December 09, 2022
Herpes virus disease is caused by herpes virinae family. It is a dsDNA virus and the family Herpesviridae can be categorized into three major subfamilies: alpha herpesvirinae, beta herpesvirinae and gamma herpesviridae. The herpes virus has unique feature: It replicates inside the host cell and produces pathogenic virions which either manifest the disease or set up latency in the host cell. The severity of pathogenesis is determined by host immune system. In immunocompromised patients, the virus reactivates again and produces a potent disease. It is a harmonious pathogen but can produce grave prognosis depending upon the host cell response. The site of latency, host range and time taken for replication is dependent upon type of herpes virus. Transmission is mainly through direct contact, no vertical transmission has been reported. Majority of herpes virus infections go unnoticed as they are asymptomatic.
Keywords:Herpes Virus; Alpha Herpes Virus; Beta Herpes Virus; Gamma Herpes Virus; Latency; Lysis; Immunodeficiency
Citation: Aditya Sharma., et al. “Herpes Virus: Immune System Mediated Latency or Lysis of Host Cell".Acta Scientific Veterinary Sciences 5.1 (2023): 55-57.
Copyright: © 2023 Aditya Sharma., et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.