Acta Scientific Microbiology

Editorial Volume 8 Issue 2

Ageing Associated Immunosenescence and Inflammageing - Cause for Greater Mortality in Infectious Diseases (Specifically Viral Diseases)

Kulvinder Kochar Kaur1*, Gautam Allahbadia2 and Mandeep Singh3

1Scientific Director, Dr Kulvinder Kaur Centre for Human Reproduction, Jalandhar, Punjab, India
2Scientific Director, Rotunda-A Centre for Human Reproduction, Mumbai, India
3Consultant Neurologist, Swami Satyan and Hospital, Jalandhar, Punjab, India

*Corresponding Author: Kulvinder Kochar Kaur, Scientific Director, Dr Kulvinder Kaur Centre for Human Reproduction, Jalandhar, Punjab, India.

Received: November 25, 2024; Published: December 01, 2024

Abstract

Ageing in variable organisms (specifically mammals) has been posited to start with encompasses nine shared properties, inclu sive of i) genomic instability, ii) telomere shortening attrition, iii) epigenetic changes, iv) decontrolling of nutrients sensing, v) mi tochondrial impairment, vi) cellular senescence, vii) changes of intercellular communications, viii)exhaustion of stem cells and ix) depletion of proteasis [1]. Immunosenescence has been thought to be an inimical event for over decades, in view of its propagating diminished capability of stimulating antibody, cellular reactions to infections along with vaccines with efficacy. Additionally, it has been thought to be an inimical event since it causes kinds of in flammation referred to as inflammageing. Recently we reviewed part of inflammageing and immunosenescence in in infectious dis eases.

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Citation

Citation: Kulvinder Kochar Kaur., et al. “Ageing Associated Immunosenescence and Inflammageing - Cause for Greater Mortality in Infectious Diseases (Specifically Viral Diseases)". Acta Scientific Microbiology 8.1 (2025): 01-04.

Copyright

Copyright: © 2025 Kulvinder Kochar Kaur., et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.




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