Attapon Cheepsattayakorn^1-4*, Ruangrong Cheepsattayakorn⁵ and Porntep Siriwanarangsun²*

¹Faculty of Medicine Vajira Hospital, Navamindradhiraj University, Bangkok, Thailand
²Faculty of Medicine, Western University, Pathumtani Province, Thailand
³10th Zonal Tuberculosis and Chest Disease Center, Chiang Mai, Thailand
⁴Department of Disease Control, Ministry of Public Health, Thailand
⁵Department of Pathology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

*Corresponding Author: Attapon Cheepsattayakorn, 10th Zonal Tuberculosis and Chest Disease Center, Chiang Mai, Thailand.

Received: February 07, 2024; Published: March 01, 2024

Abstract

Asbestos-fiber inhalation, a known cause of fibrogenic and neoplastic pulmonary injury, such as asbestosis [1,2] that triggers the development of this lung disease after more than 15 years of exposure [3-6]. These various serum biomarkers, such as 1B, interleukin (IL)-18, RANTES, tumor-necrosis-factor (TNF)-alpha [7] with sustained inflammation in non-ill-exposed-asbestos persons [3], including immune-system dysregulation, chronic inflammation, and carcinogenesis [8]. In 2020, a study was conducted in Italy compared to the period 2015-2019 demonstrated decreased Figure 1: Number of deaths involving malignant pleural mesothelioma [MPM: (A)] and asbestosis (B), by sex and age group. Italy, 2020. Deaths and the average annual number of deaths observed in 2010–2014 and 2015–2019 [9].

References

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Citation

Citation: Attapon Cheepsattayakorn., et al. “Asbestosis in the Era of COVID-19 Pandemic".Acta Scientific Microbiology 7.4 (2024): 01-02.

Copyright

Copyright: © 2024 Attapon Cheepsattayakorn., et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.