Leila Belkacemi*

Nephrology, Dialysis and Transplantation Department, CHU Nefissa-Hamoud, Algeria

*Corresponding Author: Leila Belkacemi, Nephrology, Dialysis and Transplantation Department, CHU Nefissa-Hamoud, Algeria.

Received: February 01, 2022; Published: June 01, 2022

Abstract

Phosphorus diabetes is defined by a massive tubular leakage of phosphorus leading to osteomalacia. The etiologies of this syndrome are acquired tubular leaks, hypophosphatemic rickets and oncogenic phosphorus diabetes, the latter having a benign tumor as its pathophysiology [1]; in about 90% of cases; secreting a phosphatonin most often FGF 23 which has the effect of a tubular leakage of phosphorus as well as an inhibition of the synthesis of calcitriol [2].

Keywords: Phosphorus diabetes; osteomalacia;

References

1. Véronique Breuil. “Diabètes phosphatésPhosphate diabetes”. Revue du Rhumatisme Monographies4 (2012): 253-257.
2. M Laroche., et al. “Normal FGF23 Levels in Adult Idiopathic Phosphate Diabetes”. Calcified Tissue International2 (2009): 112-117.

Citation

Citation: Leila Belkacemi. “A Rare Genetic Disease “Oncogenic Phosphorus Diabetes” Diagnosed in a Nephrological Environment: A Case Report”.Acta Scientific Medical Sciences 6.7 (2022): 15-16.

Copyright

Copyright: © 2022 Leila Belkacemi. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.