Inter-Relationship between OSA and TMD
Ruchika Sood1 and Shweta Hinduja2
1Director at Neocare Orofacial Pain Relief centre, India
2Associate oral physician at Neocare Orofacial Pain Relief centre, India
*Corresponding Author: Shweta Hinduja, Associate oral physician at Neocare
Orofacial Pain Relief Centre, India.
Received: February 23, 2023; Published: March 09, 2023
Abstract
OSA is a sleep disorder characterized by obstruction in the oro nasal airflow lasting for more than 10 seconds. The patient experiences being chocked during sleep and thereby transient awakening, which further caused reduced oxygen saturation. This partial cessation of airflow due to obstruction in the upper pharynx or nasal cavity results in snoring, known as ‘hypoaponea’, during each episode of which a reduction is respiratory effort with ≥ 4% oxygen desaturation is recorded. Each episode lasts for 10- 40 seconds, sometimes may last for one minute, during which there maybe abrupt drop in the oxygen saturation level as much as 40% in severe cases. The brain responds to each of these episodes by alerting the body and causing deep arousal from sleep that causes normal breathing, referred to as fragmented sleep quality. OSA is characterized by total number of aponea and hypoaponeas occurring per hour of sleep, which refers to/aponea/hypoaponea index (AHI) [1]. AHI is accounted for in polysomnography test which is an attended setting (sleep Laboratory) test, along with several sleep variables, such as the respiratory disturbance index (RDI). RDI is defined as sum of the aponeas, hyopapneas and abnormal respiratory events per hour of sleep [2].
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